By Eric S. Orwoll, Robert Marcus (auth.), Eric S. Orwoll MD (eds.)
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Extra info for Atlas of Osteoporosis
The allele frequencies at a polymorphism within or near the candidate gene are then compared between the two groups. Evidence of differences in allele frequencies within the two populations (association) may be the result of linkage disequilibrium with the candidate gene or possibly with another gene in close proximity; however, in practice, the candidate gene is thought likely to be the cause of the disease. Unfortunately, it is weil recognized that admixture, heterogeneity, or stratification of a population may result in significant association, even when there is no susceptibility locus in the chromosomal region .
Relation of alleles of the collagen type lalpha I gene to bone density and the risk of osteoporotic fractures in postmenopausal women. N Engl} Med 1998, 338: 1016-1021. 38. Mann V. } Clin Invest 200 I, 107:899-907. 39. : First-stage autosomal genome screen in extended pedigrees suggests genes predisposing to low bone mineral density on chromosomes I p, 2p and 4q. Eur} Hum Genet 1998, 6:151-157. 40. : A genome-wide scan for loci linked to forearm bone mineral density. Hum Genet 1999, 104:226-233.
Identification of CD36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats. Not Genet 1999,21 :76-83. 52. Wayne ML, Mclntyre LM: Combining mapping and arraying: an approach to candidate gene identification. Proc Notl Acod Sei USA 2002, 99: 14903-14906. FACTORS TRAT INFLUENCE ADULT BONE MASS Susan M. Ott A dult bone mass i5 reachcd bct\Vcen the ages of20 and 30 ycars. After that, bone loss duc TO aging is seen in both gcndcrs. In women, bone mass dccrcascs and incrcascs again wirh each rcproductivc e)'ele.
Atlas of Osteoporosis by Eric S. Orwoll, Robert Marcus (auth.), Eric S. Orwoll MD (eds.)