Read e-book online Autoimmune Diseases of the Skin Pathogenesis, Diagnosis, PDF

By Michael Hertl

ISBN-10: 3211206868

ISBN-13: 9783211206867

Univ. of Erlangen, Germany. Combines significant clinical advances within the figuring out of autoimmunity with the medical presentation and administration of issues of the surface. For clinicians and researchers. vast references, tables, figurative illustrations, and colour images are incorporated.

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2). A report by Hennekens et al. 0015). 060), respectively occurred. 44). This study was based on the selfreport of responders to a survey which had not been validated by chart review, nor had the type of implant been identified. Therefore no comment can be made regarding the risks of silicone gel breast implants. A case control study by Englert and Brooks (1994) studied 251 SSc cases and compared them with age-matched controls who were chosen at random from 28 general practitioners. 71%). 41).

Tissue Damage and Type 1 T Cells Type 1 T cells are associated with two distinct types of tissue damage. One type of tissue damage is characterized by a sterile inflammation, the other with the strong accumulation of polymorphonuclear granulocytes. Sterile type 1 responses are found in lichen planus, multiple sclerosis or autoimmune diabetes. They are associated with activated macrophages, which seem to be the effector cells of these immune responses. Like macrophages that are stimulated in vitro in the presence of IFNg, they produce large amounts of TNF, oxygen radicals, NO and other mediators of inflammation (Stenger and Modlin 1999).

Defective suppressor function of human CD4+ CD25+ regulatory T cells in autoimmune polyglandular syndrome type II. J. Exp. Med. 199, 1285–1291 Krueger, G. G, Ellis, C. N. (2003). Alefacept therapy produces remission for patients with chronic plaque psoriasis. Br. J. Dermatol. 148,784–788 Lanzavecchia, A. (1985). Antigen-specific interaction between T and B cells. Nature 314, 537–539 Leadbetter, E. , Rifkin, I. , Hohlbaum, A. , Shlomchik, M. , and Marshak-Rothstein, A.. (2002). Chromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors.

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Autoimmune Diseases of the Skin Pathogenesis, Diagnosis, Management by Michael Hertl

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